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Jun
25
2011

Skin discoloration caused by carbon monoxide poisoning – Reality vs. Holocaust eye-witness testimony

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The following text is a revised and updated version of an article originally published by the CODOH Revisionist Library website. Sensitive readers are cautioned that the article contains photographs of human corpses which may be deemed disturbing.

By Thomas Kues

1. Introduction

According to orthodox holocaust historiography, carbon monoxide from engine exhaust was used to kill nearly 2 million Jews in Poland, Serbia and on occupied Soviet territory between late 1941 and the summer of 1944. The majority of these supposed victims were allegedly killed in stationary gas chambers located in three “pure extermination camps” in the Polish General Government – Bełżec, Sobibór and Treblinka – while the remainder is said to have been killed in mobile “gas vans” that were either stationed at the Chełmno (Kulmhof) camp in the Warthegau area of occupied Poland or employed by Einsatzgruppen or SD units operating in Serbia and on occupied Soviet territory. Below is listed the victim figures for each “killing center” as currently held by the orthodox historians.

Bełżec 434,501[1]

Sobibór 170,000[2]

Treblinka 750,000-900,000[3]

Chełmno (Kulmhof) 152,000-360,000[4]

Other “gas vans” 100,000 approx.

Total: 1,606,501-1,964,501

According to most eyewitness testimony, Diesel engines from captured Soviet tanks were used as killing agents Bełżec and Treblinka, while at Sobibór, the historians claim, a petrol (gasoline) engine of unclear origin was used to produce the lethal carbon monoxide gas. As for the “gas vans” supposedly employed at Chełmno, those are commonly held to have been modified Saurer or Diamond trucks.

The danger of Diesel exhaust has long been debated by revisionist scholars. Since the early 1980s, American revisionist writer and engineer F.P. Berg has published a number of articles dealing with this issue. Their conclusion: Because Diesel engines only generate small amounts of carbon monoxide, and since Diesel exhaust contains much oxygen, the use of diesel engines as killing agents in homicidal gas chambers is preposterous. Witness testimony claiming that Diesel engines were utilized for murderous purposes are thus objectively false. To those witnesses belongs Kurt Gerstein, a certified mining engineer.

This article will not further discuss the Diesel engine issue – it will suffice to say that a number of holocaust historians cling on to the notion that Diesel engines were used for killings,[5] at least at Treblinka, while others have tried to cautiously distance themselves from the Diesel claim.[6] Instead, I will for reason of argument follow the assumption that the (hypothetical) German perpetrators used engines capable of producing lethal amounts of carbon monoxide gas. Given this, I will pose a number of questions related to the physical effects of the poison gas. How would the carbon monoxide (CO) affect the bodies of the victims? What would they look like post mortem? And, most importantly: what does the eyewitnesses to the alleged carbon monoxide gas chambers have to say about the appearance of the corpses?

2. Previous research

The main revisionist study on the issue of skin discoloration caused by carbon monoxide consists of an online article by revisionist and engineer F.P. Berg, entitled “Blue Women on the Beach – and the False Toxicity of CO2 in Diesel Exhaust”. It was written as a rebuttal to an article by Charles D. Provan, “The Blue Color of the Jewish Victims at Belzec Death Camp – and Carbon Monoxide Poisoning”, which had previously appeared in the May 2004 issue of The Revisionist. Below I will provide a summary of the relevant articles written by Berg and Provan between 1983 and 2007.

2.1. Berg’s first articles on the issue of Diesel gas chambers

The first of F.P. Berg’s writings to deal with the issue of the alleged carbon monoxide gas chambers, and especially the claim that Diesel engines were used to generate the lethal gas, was an article originally presented at the 1983 International Revisionist Conference and later, in 1984, published in The Journal for Historical Review, “The Diesel Gas Chambers: Myth Within a Myth”. In it he among other things dissected the witness account of a supposed mass gassing at Bełżec in 1942 that was left by the former SS hygiene technician Kurt Gerstein in French prison in 1945. Referring to the text of one of Kurt Gerstein’s “reports”, Berg writes:

“According to the last sentence of the text quoted, ‘the bodies were tossed out blue, wet with sweat and urine.’ Here we have a flaw as far as the death-from-carbon-monoxide theory is concerned because victims of carbon monoxide poisoning are not blue at all. On the contrary, victims of carbon monoxide poisoning are a distinctive ‘cherry red,’ or ‘pink.’ This is clearly stated in most toxicology handbooks and is probably well known to every doctor and to most, if not all, emergency medical personnel. Carbon monoxide poisoning is actually very common because of the automobile and accounts for more incidents of poison gas injury than all other gases combined.”[7]

As sources Berg gave references to two standard works on toxicology.[8] The above argument was then reiterated in a revised and expanded version of the same article which originally appeared in the revisionist anthology Grundlagen zur Zeitgeschichte (1994) under the same title and later in translation (in Germar Rudolf (ed.), Dissecting the Holocaust, Theses & Dissertations Press 2003) as “Diesel Gas Chambers: Ideal for Torture – Absurd for Murder”. In this appearance the above quoted passage was complemented with a further reference to recently published specialist literature.[9]

2.2. The 2004 article by Charles D. Provan

In his article The Blue Color of the Jewish Victims at Belzec Death Camp – and Carbon Monoxide Poisoning[10] Provan asserts that bluish color or bluish tinge attributed to the Bełżec victims by Gerstein and later Pfannenstiel can be explained as cyanosis. “Blue”, Provan writes, “is a regular (and documented) color for carbon monoxide poisoning, especially when the victims are alive, but also when the victims are dead.” In regard to fatal cases of CO poisoning, Provan quotes a number of studies indicating that “in some cases” of fatal poisoning there is “no cherry-red coloring of the skin”, that in some cases the appearance of the victim is instead “cyanotic”, and that the cherry-red discoloration might be “slight” due to low saturation (i.e. low carboxyhemoglobinal level) and in some cases obscured because of “associated cyanosis”. Provan takes the above as evidence that what Gerstein and Pfannenstiel said in regards to the color of the corpses is “possible”, and that Berg in his previous articles had reached the wrong conclusions.

2.3. The rebuttal of F.P. Berg

Berg opens his rebuttal to Provan[11] stating that the assertion of blue corpses “is totally at odds with the claims (…) that the toxic ingredient [in the exhaust gas used as the killing agent] was carbon monoxide.” The texts on cyanosis referenced by Provan, Berg notes, “fail to use the words “blue” or even “bluish” at all”. “The simple fact”, Berg further contends, “is that the blue appearance of “cyanosis” does not correspond at all to the general “blue” appearance of the “blue corpses” that Gerstein or Pfannenstiel allegedly saw (…)”. Corpses may be multi-colored, and thus “blue” cyanosis may appear on one part of the body, while the rest of it displays a cherry-red color. Cyanosis occurring in connection with carbon monoxide poisoning is “associated” with the poisoning and not in itself a product of any reaction between carbon monoxide and the victim’s blood. Reactions of carbon monoxide with blood are more or less bright red, never blue. Provan is wrong in defining cyanosis as a “medical term for blue coloring occurring in a patient or corpse” since “cyanotis” is not simply the medical term for blue coloring, but only applies to some varieties of blue discoloration. One would not be able to conclude a case of CO poisoning from the mere presence of cyanosis; the color of the victim’s blood would also be examined.

While cyanosis may appear in some fatal cases, “the appearance of a generally “blue” corpse is extremely rare if it ever occurs at all” (Berg). Below a carboxyhemoglobin level of 30% a living body or corpse may indeed display cyanosis without accompanying bright red discoloration, but as the lethal level for most individuals lies around 60%, an overwhelming majority of corpses would definitely show some nuance of red. Variations and exceptions to this occur in only around 6% of all cases. Also, the reddish color when occurring “tends to be extremely intense and dramatic whereas cyanosis is an extremely subtle coloring in which most of the skin is merely pale” (Berg). A lay observer would thus have a hard time noticing any cyanotic cases, whereas the red discolored corpses would be immediately noticeable. “There is good reason to believe”, Berg writes, “that a cyanotic description in our context does not really mean blue at all — but merely blue by contrast or in comparison to other parts of the same or other bodies.” In regards to the Pfannenstiel testimony, Berg remarks that Pfannenstiel “noticed nothing special about the corpses” except for a bluish tinge to the face of some of them, and that no mention of any red discoloration is made, two things which combined speaks against the reliability of this witness. Berg also strongly criticizes Provan’s way of mixing fatal and non-fatal cases of poisoning, as well as “immediate” fatal cases with “delayed” ones. Living victims of CO poisoning may be partially cyanotic and partially red (with a “flushed” or pink appearance) or cyanotic with only negligible or unnoticeable red discolorations. Dead CO victims on the other hand are usually red or cherry-red. In the rare cases (around 9% of all cases) when cyanosis appears associated with fatal CO poisoning, it tends to be appear restricted to parts of the body where the skin is more translucent, such as the lips or nasal openings. The alleged observations of Gerstein and Pfannenstiel are thus not reconcilable with known medical facts.

3. The difference between fatal and non-fatal cases of CO poisoning

In discussing the issue of discolorations in the skin of CO gassing victims, it is important to note the difference between fatal and non-fatal (i.e. clinical) cases of CO poisoning. In the writings of anti-revisionists, we often find quotes from medical literature such as:

The classic findings of cherry-red lips, cyanosis, and retinal hemorrhages occur rarely.[12]

Or:

The classic ‘cherry-red’ skin coloration is actually rare, and patients are more likely to appear pale or cyanotic.[13]

As F.P. Berg points out, statements such those above appears to refer mainly to clinical cases of carbon monoxide poisoning, i.e. cases where the poisoned person was found alive and received treatment before he or she either survived, or died (therefore the word “patients” in the second quote). A statement similar to the ones quoted above can be found in the standard work A guide to general toxicology (1983):

Carbon monoxide poisoning may result in blisters or bullae over pressure areas but the classic cherry red color of the skin is rare.[14]

When, however, the text within which this quote appears is read more closely, it becomes evident that the author(s), without stating this explicitly, is referring mainly or even exclusively to clinical cases.[15] In fact, specialist literature on toxicology and emergency medicine by its very nature normally focus on clinical cases, while cases involving untreated fatal cases are normally treated in writings related to forensic medicine.[16] An article from 2007 authored by Nicholas Bateman, a professor in clinical toxicology, indirectly confirms that deep red or “cherry pink” discoloration is rare among surviving victims, but more common in fatal cases (emphasis added):

Skin blistering may occur if the patient lies unconscious for some hours before being discovered, and the skin is more likely to be cyanosed than to have the cherry-pink colour that is described to be a classical feature of CO poisoning, but rarely seen in living patients.[17]

The letter by Bruno Simini to The Lancet, often cited by anti-revisionists, in which it is stated that “cherry-red discoloration in CO poisoning is quite rare” and that “most doctors overestimate the frequency of cherry-red discoloration in CO poisoning” is also clearly referring to clinical cases of poisoning, since it only refers to “surveys of patients” i.e. treated victims of CO poisoning.[18]

The case reports and medical papers which I quote and refer to in the next section clearly proves that deep red or cherry red discoloration of the skin is virtually always present among fatal cases of CO poisoning. In the section after that I will contrast the contents of the medical case reports and findings with statements made by professed eyewitnesses to the alleged homicidal gas chambers and “gas vans”.

Illustration 1: Reddish flush in a non-fatal case of CO poisoning.[19]

 

Illustration 2: Typical red discoloration in victim of fatal CO poisoning.[20]

 

Illustration 3: A fatal case of CO poisoning displaying distinctive reddish-pink discoloration.[21]

 

Illustration 4: Bright red lividity in a victim of CO poisoning.[22]

4. Verified cases of discoloration resulting from carbon monoxide poisoning

Below I will provide brief summaries of a number of case reports and medical papers concerned with skin discoloration as an effect of CO poisoning.

Item 1: The man with the red face

The following case from mid-60’s America involved the suicide attempt of a 21-year old white male of Italian descent:[23]

When seen on the morning following his admission the author was struck by the appearance of the patient’s cherry-red face. Additionally, he was thick-tongued in speech, lethargic and showed impairment of orientation as regards time and place. Confusion as to what had brought about his admission was noted.

The writer’s initial impression was acute brain syndrome but one whose etiology might involve carbon monoxide poisoning. Thus, the patient was questioned closely as regards the circumstances and details of his suicide attempt. Elicited from the patient were additional facts that he had fallen asleep in his car with the engine running and the windows closed. Twelve hours later, he awoke and returned home to tell his parents what he had done. At that time his clothes were covered by vomitus. It became apparent that a most important clinical sign and area of history had been over-looked previously.”

Thus it is apparent that cherry-red skin discoloration can be highly visible even among survivors of carbon monoxide poisoning. Red discoloration of the skin is thus not limited to the lividity of fresh corpses, but appears in the still living victim’s body as the mechanical result of carbon monoxide being absorbed by the bloodstream. This is because, as F.P. Berg writes in his rebuttal to Provan, “when carbon monoxide reacts with human blood, it forms carboxyhemoglobin which above concentrations of 30% is a bright red, becoming brighter and more intense as the concentration increases”, that is, the discoloration begins immediately with the reaction of the blood with the CO, and is then increased by the inflow of CO. Following death the discoloration is then concentrated by the pooling of blood that is livor mortis (post-mortem lividity).

Item 2: A dead girl in Italy

This case involved a 21 year old white female found dead in a country house owned by her family. It was later determined that her death had been unintentionally caused by a gas water heater. We are told by the authors of the case report that “[t]he pale cherry pink colour of the victim immediately suggested a carbon monoxide poisoning.” A spectrophotometric measurement of the blood showed a carboxyhemoglobin level of 60%. The report also mentions that among survivors of CO poisoning, the mean carboxyhemoglobin level is 28.1%, while among fatalities the mean level is 62.3%. At a level of 50%, the probability of survival is more or less 50%.[24]

Item 3: A German report on six “unusual” cases of fatal CO poisoning

This article[25] states that, despite the presence of indicative death scenes and/or characteristic findings of the external (coroners’) examination, about 40% of all unintentional fatal cases of carbon monoxide poisoning remain unrecognized until the autopsy. To illustrate possible reasons for this, the authors describe six individual cases. In case 1 and 2, involving a middle-aged couple, the bodies were found in a state of extreme putrefaction, so that the cause of death could only be recognized through spectrophotometrically analyzing the carboxyhemoglobin level of the oedema fluid that had gathered in the scalps of the victims. Case 3 involved a young truck driver, found dead in the closed cab of his vehicle and not displaying any clear external signs of CO poisoning, despite a carboxyhemoglobin level of 83%. Case 4 involved a 19 year old male found dead in a flat. Despite a carboxyhemoglobin level of 65% his body lacked “the bright pink coloration of livor mortis”. Case 5 involved a 27 year old male discovered dead in his flat with a carboxyhemoglobin level of 80%. His body was found in a state of advanced decomposition. Case 6 involved a 42 year old female found dead in the garage beside her car. The body did not show any clear external signs of CO poisoning despite a carboxyhemoglobin level of 46%. As stated already by the title of this article (“Unusual carbon monoxide poisoning”) these six cases (in particular cases 3, 4 and 6) are to be viewed as anomalous.

Item 4: An American case of CO poisoning without cherry-red discoloration

According to the authors of this article, carbon monoxide poisoning “typically causes so-called cherry-red livor of the skin and viscera.” They then report of a case of CO poisoning in which this cherry-red livor did not develop. It involved a 75 year old white male found dead in his car during a cold winter. His carboxyhemoglobin level was measured as 86%. The authors inform us that “the curious absence of cherry-red livor” was studied and the decedent’s tissue and blood specimens tested at various temperatures. The tests showed that neither the blood nor the tissue of the victim had a tendency to develop cherry-red color, regardless of temperature.[26]

Item 5: An optical study of discolorations

In this South African study of 10 fatal cases of carbon monoxide poisoning, the skin color of the victims’ bodies was analyzed by the help of reflectance spectrophotometry, with the values converted to visual equivalents. It was found that several circumstances contribute to the difficulty of identifying the cherry-red color in the skin, among them low CO concentration in the blood, skin pigmentation, washing-out of previously high CO concentrations, and deep venous dilatation combined with superficial vasoconstriction (narrowing of the blood vessels), producing the impression of cyanosis. It was further found that the color of the altered blood “depends on the way the red cells are massed together, their depths below the surface, and the brightness of the background against which they are viewed.”[27]

Item 6: A study of 15 CO victims at an Indian hospital

This study, published in 2001, was carried out at a hospital in a provincial Indian city which is located on an altitude of 5000 ft above mean sea level. It involved findings in 40 cases of accidental carbon monoxide poisoning, 25 of the clinical, 15 of them post mortem. The autopsy findings revealed “deep red discoloration of skin and serous membranes” in 12 of the 15 corpses.[28] This study is important for the topic of the present article, since it shows that deep red discoloration is displayed by a majority of victims of lethal carbon monoxide poisoning even when the skin of the victims are of a darker pigmentation than the average Caucasian’s.

Item 7: An Austrian study on 182 cases of fatal CO poisoning

This study[29] consists of an analysis of autopsy reports of postmortems performed at the Viennese Institute of Forensic Medicine between 1984 and 1993. The aim of this survey was to determine whether the cherry-pink coloring of livor mortis[30] is a reliable finding for the coroner to suspect a carbon monoxide-related death immediately at the death scene. It involved 182 cases of unintentional carbon monoxide-related deaths: 92 females and 90 males. The authors found a strong association between the carboxyhemoglobin level (i.e. the level of CO concentration in the blood’s hemoglobin) and the cherry-pink coloring of livor mortis: “in 98.4% of unintentional carbon monoxide-related deaths livor mortis was clearly cherry-pink.”[31] It was determined that fresh corpses with carboxyhemoglobin levels greater than 31% show “a clear cherry-pink coloring of livor mortis.”[32] The survey further indicated that the Viennese coroners’ inability to recognize cases of unintentional carbon monoxide fatalities immediately at the death scene was correlated to the age of the victim: the older the victim, the worse the coroner’s recognition.

In conclusion, the authors of the article suggest that coroners should be recommended to examine naked corpses thoroughly, and especially the color of livor mortis. In this way, they write, a carbon monoxide-related death can be recognized immediately and the source of the gas release identified, thus protecting other people from the risk of poisoning.

Item 8: A survey of 388 car exhaust gas suicides in Denmark 1995-1999

This study[33] from 2005 consists of a survey of 388 cases of suicide by means of engine exhaust gas carried out in Denmark between 1995 and 1998. Of the suicides 343 were males and 45 females. It was found that in 11 cases (2.8%) putrefaction or burns were so extensive that livor mortis could not be found, while “the characteristic pink livor mortis” was found in 353 cases (91% of the total cases, 93.6% of those with livor mortis). Only in 9 cases (2.4% of those with livor mortis) did the victims show a normal-colored livor mortis. In 3 of those 9 cases the victim had survived more than a day after the poisoning, implying a positive correlation between the cherry-red discoloration of livor mortis and the carboxyhemoglobin level. In 15 cases the author of the autopsy report had neglected to write down the color of livor mortis.

Summary of the medical evidence

From the above summarized cases we may conclude that:

  • Cherry-red discoloration sometimes appears in non-fatal cases of CO poisoning, i.e. it is visible also in ante-mortem states (Item 1). According to available medical literature, such cases are not the rule, but on the other hand not highly exceptional. Such discoloration would appear more or less directly after the blood cells had started absorbed the carbon monoxide. The visibility of the deep red discoloration is related to the concentrations of CO in the blood (i.e. the carboxyhemoglobin level), as well as other factors such as pigmentation (Item 5). In the case of the alleged gas chamber victims it is reasonable to assume that their carboxyhemoglobin level would be much higher than that of the average CO poisoning survivor (that is 28.1%, whereas in fatal cases the concentration averages 62.3%; cf. Item 2), thus greatly increasing the number of individual cases with cherry-red discoloration appearing already ante-mortem or prior to the onset of livor mortis.
  • According to Item 7 fresh corpses with carboxyhemoglobin levels greater than 31% shows clear discoloration. This level is only 2.9% above that of the average survivor of CO poisoning (cf. Item 2).
  • In cases of fatal CO poisoning, deep red discoloration of the livor mortis is visible in many cases even when the victim’s pigmentation is much darker than that of the average Caucasian (Item 6).
  • In fatal cases of CO poisoning, absence of cherry-red lividity is regarded as “curious” or “unusual”. Individuals whose blood and tissue lacks the tendency to develop the cherry-red color are very much an exception (Item 4). In many of the fatal cases where discoloration could not be detected, this was due to the corpse having entered the stage of advanced decomposition, or from having suffered severe burns (Items 3, 8).
  • Deep red/cherry-red discoloration of livor mortis is present in at least 95% of all fatal cases of carbon monoxide poisoning (Items 7 and 8).

5. Eyewitness descriptions of alleged carbon monoxide victims at Bełżec, Sobibór, Treblinka, and Chełmno

Witness 1: Kurt Gerstein

As a captive of Allied forces in France, former SS hygiene technician Kurt Gerstein wrote a number of reports in which he claimed to have witnessed a mass gassing at Bełżec in August 1942. In the two reports indisputably written by Gerstein in French on April 26, 1945, the bodies of the gassing victims are described in the following way:

The blue bodies are thrown, damp with sweat and with urine, the legs full of excrement and menstrual blood.”[34]

In the German-language Gerstein reports which Henri Roques designate T III and T VI the word “blue” is not present. It is likewise not present in the French text T Va, dated to May 6, 1945. The German text T IV contains no corresponding passage.

Regarding the blueness of the Bełżec corpses and the issue of cyanosis, see Section 2 above.

Witness 2: Wilhelm Pfannenstiel

The professor of hygiene at the University of Marburg-Lahn Dr. Wilhelm Pfannenstiel allegedly accompanied the aforementioned Kurt Gerstein on his trip to Bełżec in August 1942. After the war Pfannenstiel was arrested but never sentenced to prison. Instead he was on a number of occasions summoned as a witness for the prosecution in trials dealing with the alleged homicidal gas chambers at the Reinhardt camps. In 1950 he testified before a court in the German city of Darmstadt:

I noticed nothing special about the corpses, except that some of them showed a bluish puffiness about the face. But this is not surprising since they had died of asphyxiation.”[35]

Since Pfannenstiel was without question familiar with the texts of the Gerstein reports, it is fully possible that he also derived his description of the corpses from one of the two French texts. As an alternative, it cannot be excluded that Pfannenstiel, with his thorough background in medicine and hygiene studies, was familiar with asphyxiation symptoms and thus also able to fabricate a vague description with the ring of authority. As for the Pfannenstiel testimony I once again refer to Berg’s article summarized above.

Witness 3: Karl Alfred Schluch

SS-Unterscharführer Karl Alfred Schluch was posted at Bełżec from June 1942 until early summer 1943. His work at the camp up until December 1942 supposedly involved accompanying the naked Jewish victims through the camouflaged “sluice” which led to the gas chambers. Schluch was acquitted at the trial of former Bełżec camp staff held in Munich in 1963. In connection with this trial the witness made the following statement regarding the bodies of the gas chamber victims:

The corpses were at least partially besmirched with excrement and urine, others in part with saliva. The lips and nose tips of some of the corpses had turned blue. With some the eyes were closed, with others the eyes had rolled.”[36]

Now it is possible that the lips, and possibly also the nose tips, of carbon monoxide victims would look purple-bluish as a result of cyanosis. The problem is that this is the only kind of discoloration that the witness claims to have been aware of. Are we to believe that Schluch noticed a few purple-bluish lips, but completely missed the large red discolorations?

Witness 4: Adolf Eichmann

Adolf Eichmann testified during his trial in Jerusalem that he had visited three camps were carbon monoxide was allegedly used to exterminate Jews: Chełmno (Kulmhof), Treblinka, and an unidentified camp in the Lublin area commonly assumed to have been Bełżec. Only in regard to the first camp does Eichmann claim to have witnessed the bodies of the alleged victims. This is how Eichmann described the murder of Jews in “gas vans” at Chełmno:

I went myself to a small wood and just as I got there the omnibus also arrived, it pulled up beside a pit which had been dug up, the doors were opened and out of them poured corpses, down into the pit. One upon the other. It was a ghastly inferno. No, a super-inferno. To me they looked as if they were still alive. But now each and all of them were dead.”[37]

Thus according to Eichmann the corpses of the victims looked the same way as when they had been alive. The vagueness of the description makes the testimony weak evidence in any case, but it might be safely assumed that Eichmann would have noticed and remembered large red discolorations on the corpses from the gas vans, if he had in fact seen any.

Witness 5: “Szlamek”

This key witness to the alleged gas van mass murders in Chełmno, who has been identified as either a certain Jakov Grojanowski or Szlojme Fajner, claims the following in his testimony, reportedly dating from February 1942:

How did the corpses appear? They were not burned, not black. The complexion of their faces was unchanged. Almost all the dead were lying in their excrement.

[...].

It seemed that they had only been put to sleep; their cheeks were pale and they kept their natural skin color.”[38]

Thus the corpses displayed no skin discoloration whatsoever.

Witness 6: Rudolf Reder

The witness Rudolf Reder, born in 1881, is supposed to have spent a significant portion of his nearly four month long stay at Bełżec dragging corpses from the camp’s alleged gas chambers to massive burial pits. On December 29, 1945, Reder was interrogated by the Polish Judge Jan Sehn. Regarding the physical appearance of the gas chamber victims, the witness stated:

I was often on the ramp at the moment the doors were opened, but I never smelled any odor, and on entering a chamber right after the doors were opened I never felt any ill effects on my health. The bodies in the chamber did not show any unnatural discoloration. They looked like live persons, most had their eyes open.”[39]

The Bełżec key witness Reder is thus clearly of the opinion that the gassing victims displayed no cherry-red discoloration.

Witness 7: Eliahu Rosenberg

The Jewish witness Eliahu (Elias) Rosenberg supposedly spent several months working in close proximity of the alleged Treblinka gas chambers,[40] dragging thousands of corpses from the “death chambers” to mass graves. In a 12-page typewritten deposition in German which Rosenberg left in Vienna on December 24, 1947, the appearance of the gas chamber victims is described thus:

The corpses were very bloated, their skin looked gray-white and easily peeled off, so that it hung from them like shreds. Their eyes protruded and the tongues hung out of their mouths.”[41]

Rosenberg’s description of the hue of the corpses is clearly not consistent with the red discoloration resulting from carbon monoxide poisoning.

In addition to Rosenberg, the Jewish writer Rachel Auerbach states in her essay “In the Fields of Treblinka” from 1946 that “the bodies were naked; some of them were white, others were blue and bloated.”[42] Auerbach had not herself been interned at Treblinka, but visited the remains of the camp in 1945 as part of an official inspection tour. Her essay is reportedly based on written testimony and talks she had with former Treblinka inmates. Another secondary account derives from the writings of a certain Jacob Mittelberg, who spent only a few hours in Treblinka before being transferred to Majdanek. Mittelberg visited the site of the “death camp” after the war in the company of Rachel Auerbach and a number of former Treblinka inmates, who told him that “when the doors of the gas chambers were opened, the people were blue and so pressed together as to be unrecognizable.”[43] Soviet-Jewish propagandist Vasily Grossman wrote in 1945 after his visit to the former camp site that “People who were unloading the chambers told me that the faces of dead were very yellow”.[44]

Witness 8: Theodor Friedrich Leidig

As far as I have been able to determine the only eyewitness to an alleged mass murder with exhaust gas to have spoken of corpses with red or reddish coloring was a certain Dr. Theodor Friedrich Leidig of the Kriminaltechnisches Institut (KTI) of the RSHA. Dr. Leidig claimed to have witnessed the murder of Russian POW’s detained at Sachsenhausen using a “gas van”:[45]

We then went to another place, where we once again encountered the van. It turned out that we were now at the crematorium. I still remember that one could look through a peephole or a small window [Scheibe] into the interior of the van, which was illuminated.

One could see that the people were dead. Then the van was opened. Some corpses fell out, the rest were unloaded by prisoners. The corpses had, as was determined by us chemists, the pinkish-red [rosa-rote] appearance which is typical for people who have died from carbon monoxide poisoning.”

Regardless whether this testimony is reliable or not – and we have in fact not a single shred of documentary or technical evidence that supports it – the following observation is inevitable: Leidig clearly knew from his studies that humans who have died of CO poisoning ought to look “pinkish-red”, so in case he was forced or felt impelled to make up a false story, he would have little problem making it a plausible-sounding one. A testimony from a layman mentioning the presence of reddish-pink discoloration would clearly be of a higher evidentiary value, as the possibility that the witness had drawn from a priori knowledge to embellish his story would be much smaller.

6. Rebuttals to possible counter-arguments

Below I will discuss four possible counter-arguments which may be raised against the revisionist critique of the eye-witness testimony.

Argument 1: The studies cited by revisionists are irrelevant because they refer to livor mortis

As has been explained above, the cherry-red discoloration appears as a mechanical effect soon as the carbon monoxide has been absorbed by the blood cells and is thus visible on post-mortem bodies (especially pronounced in the livor mortis, as during this phase the blood is concentrated due to gravity-induced pooling) as well as in ante-mortem states (to a variable degree) and even in some cases where decomposition has already set in. The medical studies and case reports quoted in this article and others are therefore relevant, whether referring to livor mortis or ante-mortem appearances of red discoloration.

Argument 2: Most or all of the victims were deeply anemic, something which would have prevented visible discoloration from ocurring

Anemia is medically defined as a qualitative or quantitative deficiency of hemoglobin, the molecule found inside red blood cells which causes the blood to look red. Anemia results either from excessive blood loss (due to hemorrhage or chronic loss of smaller volumes of blood), excessive destruction of blood cells, or a deficient production of new red blood cell. The idea of the counter-argument is that severe anemia would prevent the red discoloration from appearing in the gassing victims.

In the case of the Jewish deportees, anemia might have been caused either by inadequate intake of vitamin B12 and/or folic acid (leading to macrocytic anemia), or by iron deficiency (causing microcytic anemia). While mild anemia caused by iron deficiency among women of childbearing age is not uncommon even in the western world of today, it is very rare among men and children.

How common then was anemia among the populations of the wartime Jewish ghettos of Poland, where malnutrition, starvation and epidemics indeed took a heavy toll on the inhabitants? This question is very difficult to give a definitive answer to, but a number of indications may be gleaned from the book Hunger Disease. Studies by the Jewish Physicians in the Warsaw Ghetto, edited the former Director of the Columbia University Institute of Human Nutrition, Dr. Myron Winnick.[46] In this volume, Winick presents a report on nutrition-related diseases prepared by a group of Jewish physicians in the Warsaw Ghetto between 1940 and 1942. The group, led by Dr. Israel Milejkowski, worked out the details of the study in secret meetings, had medical equipment smuggled into the ghetto, and later smuggled the finished manuscript out of it. The small team of 28 Jewish medical experts included Dr. Mieczyslaw Kocen, a specialist in blood diseases who himself was later allegedly exterminated at Treblinka. The manuscript of the report, which escaped the war tumult relatively unscathed, was published in limited Polish and French editions by the American Joint Distribution Committee. It remained most obscure however, until it surfaced in the United States in the late 1970s and was published in edited form by the abovementioned Winnick.[47]

Regarding the changes of blood characteristics in hunger disease victims the ghetto physicians noted the following:

Red blood cells examined in 80 cases decreased from 3 million per cubic millimeter to between 1.5 and 1 million and in some cases even below. Hemoglobin decreased to 60 to 70% and in some cases ranged as low as 10%. Color index was usually 1 or less, and rarely reached 1.15. Examining a drop of fresh blood we noticed that the red blood cells do not aggregate normally into rolls but remain single or group into small clusters. Anisocytosis and even more often microcytosis are present, macrocytosis is rare, and there are no nucleated red blood cells. Often the red blood cells are colorless and irregularly shaped. These are symptoms of hypochromic anemia in the recovery phase as indicated by a high percentage of reticulocytes.”[48]

The Warsaw doctors pointed out that “hunger disease” does not result in a decrease of the blood volume of the victim:

In cachexia and hunger edema there is no anemia in the strict sense because blood volume is not decreased in proportion to body weight. Since there is a low percentage of red blood cells in a drop of blood, this would be classified as normovolemic oligocytemia.”[49]

Rather than a decrease of the total number of red blood cells, “hunger disease” tends to cause a dilution of the blood through the increase of the water content:

In normal specimens plasma contains 89 to 90% water and red blood cells contain 63 to 67% water. In our patients’ specimens plasma contained 93 to 94% water and red blood cells only 58%.

The changes described in the water content of the blood can produce a pseudoanemia in patients with cachexia or hunger edema. The dryness of the red blood cells explains the presence of microcytosis.”[50]

In a study of child victims of hunger disease it was observed:

Anemia was usually mild (3 to 3.5 million red blood cells, but sometimes under 2 million, or color index about 1). Even in advanced anemia no young red blood cells were found. In evaluating the degree of anemia, we had to consider “blood dilution,” which was present in every case of severe malnutrition, even the dry form without edema. (…) Dr. Apfelbaum’s research on the volume of blood in adults suffering from hunger disease has demonstrated an increase in blood volume per kilogram of body weight. This factor must also be considered in evaluating the degree of anemia.”[51]

On the subject of child victims of malnutrition, Winnick comments:

One might assume that since these children, especially the older ones, were reasonably well nourished before the war (unlike most children in developing countries) they had built up significant reserves of vitamin A prior to contracting hunger disease. (…). Finally, vitamin A requirements, like those for other vitamins, might decrease during semistarvation.”[52]

Winnick further notes:

This results not only in hemodilution which, as we shall see, contributed to the anemia and leukopenia reported in the next chapter, but also in a reduction in the efficiency of the blood as a carrier of nutrients. Thus the vascular system is forced to supply more of the ‘poorly nourished’ blood to the ‘hungry’ tissues and organs. The absolute anemia (…) reduces the amount of oxygen carried by the blood and again increases the total blood requirements of the tissues even though they are consuming less oxygen.”[53]

Another study of the Warsaw physicians showed that some degree of anemia was common among patients of hunger disease but that

of 32 cases only six had 4 to 5 million red blood cells. Thus anemia was prevalent. The largest group of people had 3 to 4 million blood cells. Therefore we consider this number as average for slightly advanced hunger disease.”[54]

However, according to the table following this paragraph 10 of the cases displayed a level of 3-4 million red blood cells per cubic millimeter, while 9 cases displayed a level of 2 million or less. Thus only a minority of the studied cases suffered from what could be defined as severe anemia. Further among the conclusions we read that

Anemia is normochromic or hyperchromic and only very rarely hypochromic. There is anisocytosis with a predominance of macrocytes.”[55]

Winnick summarizes the post-mortem case studies performed as follows:

They [the physicians] report on 492 autopsies performed in the 2 ½ years that preceded the deportations. These were cases of ‘pure’ hunger disease with no other complications. This represented about 15% of the total number of autopsies performed in their departments during the same period. They divided their material into four periods beginning in January 1940 and ending on July 22, 1942, and point out that the number of cases of hunger disease increased with time.”[56]

In a series of tables the Warsaw physicians list the following gross changes in the “hunger disease” victims:[57]

1. Pale cadaver-like skin in 82.5% of the cases. Dark brown-colored skin in 17.5%.

2. Edema in one third of the cases. Effusions were most frequent in the abdominal cavity when they occurred.

3. Edema was rare in cases of “brown skin,” whereas the pale skin group had either the edematous or the dry form of the disease.

4. Severe atrophy occurred in heart, liver, spleen, and kidney.

5. Brain weight remained unchanged (these were adult patients).

6. Marked skeletal muscle atrophy.

7. Edema of the small intestinal wall with swollen reddish discolored mucosa and mucus appeared in 27.2% of the cases.

8. Thin watery bile in 77.7% of the cases.

9. Reduced number of fat bodies in the adrenals in 50% of cases.

10. Jellylike consistency in bone marrow of certain cases.

11. Emphysema in 13.8% of cases.

12. Anemia in only 5.5% of cases.

13. Almost 50% of the cases had intestinal changes that could be classified as pseudodysentery. An equal number of these cases fell into the edematous and nonedematous groups.

The above can be taken as a strong indication that even among fatal cases of malnutrition, anemia was far from always present. Even if no definitive answers may found in regards to this question, it seems far-flung to assert that a majority of the Jewish deportees who arrived at Treblinka were afflicted with anemia severe enough to prevent the appearance of a visible livor mortis or other variants of skin discoloration.

Argument 3: The lighting may not have been adequate for the eye-witnesses to see the colors of the corpses properly

This argument is easily dismissed. Rosenberg and Reder claims to have worked not only with removing the corpses from the gas chambers, but also with transporting them to the mass graves. It is generally asserted by holocaust historians that this activity was mainly carried out during the day,[58] so that in most if not all cases the Arbeitsjuden engaged in the corpse-dragging must have been able to observe their macabre burden in full daylight.

Argument 4: The inmates working with transporting the corpses might not have noticed the color of the livor mortis since it would have appeared on the half of the bodied turned towards the ground

There are two obstacles to this argument. On its way from the gas chamber to its final place in one of the mass graves the corpse would have made at least two stops, first close to the gas chambers, where the “dentists” would check its teeth and pull out any gold present, the second at the edge of the burial pit, where it had to be arranged with the other bodies in some fashion. In order to efficiently arrange the huge number of bodies in the mass graves, a portion of them would most likely have had to be turned around. In any case it seems logical to assume that a great many of the hypothetical gassing victims would have been turned over at least once on their way to the burial pits. That the inmates who worked day after day with these routines would have managed to completely miss the large, brightly discoloured portions of skin is simply out of the question – unless we assume that the clever Nazis selected only colorblind Jews for these work commandos!

7. Conclusion

In a medical article from 2004 we find the following stated regarding the appearance of cherry red skin discoloration in cases of carbon monoxide poisoning:

The classical cherry red appearance is not seen in all cases of acute poisoning, and may not be apparent even in cases of severe toxicity.[59]

However, in the case of the alleged mass gassings at the Aktion Reinhardt camps and Chełmno, all of the (alleged) victims can safely be regarded as victims of acute poisoning, and since the witnesses to the alleged gassings supposedly observed – often at very closely distance – not only one or two corpses, but hundreds, thousands, even tens of thousands of corpses, it natural follows that witnesses such as Reder, “Szlamek” and Rosenberg would have observed a very large number of bodies showing cherry red discoloration. That not a single one of the alleged eye-witnesses to mass gassings at the above listed camps mention the highly eye-catching type of discoloration that most often accompany lethal carbon monoxide poisoning is in itself enough to throw doubt upon the alleged truthfulness of their statements.[60] The apparently isolated case of Theodor Friedrich Leidig , not only because of his background but also due to the fact that he describes something not part of the holocaust per se, namely the (alleged) murder of a group of Russian prisoners of war at an “ordinary” concentration camp. When key witnesses from the “extermination camps”, however, reports the corpses to have been blue, white, grayish, or even without any discoloration whatsoever, then we can be certain that something is not right with their gas chamber testimonies.


[1] The Bełżec camp was opened in March 1942, ceased operating in late November or early December 1942 the same year, and was fully dismantled during the following year. The so-called Hoefle telegram, discovered in 2000 by historians Peter Witte and Stephen Tyas, shows the number of Jews deported to the Reinhardt camps up until December 31, 1942. The total stated for Bełżec is 434,508. It is alleged by historians that merely 7 Jewish prisoners managed to escape from the camp (cf. Carlo Mattogno, Belzec in Propaganda, Testimonies, Archeological Research, and History, Theses & Dissertations Press, Chicago 2004, p. 51) – I have subtracted this number from the total.

[2] In his study on this camp Jules Schelvis makes a convincing case that at the most 171,000 Jews were deported to this camp; of these at least 1,000 Jews (among them Schelvis himself) were selected for work in nearby labor camps; J. Schelvis, Sobibór. A History of a Nazi Death Camp, Berg Publishers/USHMM, Oxford 2006, p. 110, 198).

[3] 750,000 is the figure championed by Raul Hilberg in the “definitive” 2003 revised edition of his standard work The Destruction of the European Jews, while the 900,000 figure is advanced by German historian and court expert Wolfgang Scheffler (cf. Adalbert Rückerl, NS-Vernichtungslager im Spiegel deutscher Strafprozesse, dtv, Frankfurt 1977, p. 199). From the aforementioned Hoefle telegram we know that a total of 713,555 Jewish prisoners were sent to Treblinka during 1942. As all sources agree that the number of transports to Treblinka in 1943 was much lower than in the previous year, and that there were long periods without any convoys arriving, it is unlikely that the total number of arrivals exceeded 800,000.

[4] According to the Korherr report 145,301 Jews “were moved through the camps in the Warthegau” (it is apparent that Korherr here made a mistake in writing the plural camps). Orthodox historians maintain that Chełmno, which ceased receiving transports in late 1942, reopened in the summer of 1944 and was used again to murder a number of convoys from the Łódz ghetto; thus the lower victim estimate of 152,000 (cf. Israel Gutman (ed.), Enzyklopädie des Holocaust, Argon Verlag, Berlin 1993, vol. I, p. 280). As shown by Carlo Mattogno, however, it is dubious that these second phase transports to the camp actually took place (cf. C. Mattogno, Il Campo di Chełmno tra Storia e Propaganda, Effepi, Genua 2009, chapter 13). The higher figure of 360,000 is taken from Martin Gilberg, Endlösung. Die Vertreibung und Vernichtung der Juden. Ein Atlas, Reinbek, Rowohlt 1982, p. 169. At the International Military Trial at Nuremberg it was claimed that 340,000 Jews had been killed at Chełmno (IMT, Vol. VIII, p. 364).

[5] Cf. Richard Evans, The Third Reich at War, Penguin Books, London 2009, p. 290, 292; Peter Black, “Foot Soldiers of the Final Solution: The Trawniki Training Camp and Operation Reinhard”, Holocaust and Genocide Studies, vol. 25, no. 1 (Spring 2011), p. 20, 32.

[6] Cf. Achim Trunk, who in his essay “Die todbringenden Gase” (in: Günter Morsch, Betrand Perz (eds.), Neue Studien zu nationalsozialistischen Massentötungen durch Giftgas. Historische Bedeutung, technische Entwicklung, revisionistische Leugnung, Metropol Verlag, Berlin 2011)  writes: “In the case Diesel engines were utilized, death certainly took much longer to occur, as Diesel machines produce considerably less carbon monoxide” (“Falls Dieselmotoren eingesetzt wurden, dauerte das Sterben mit Sicherheit sehr viel länger, da Dieselmaschinen deutlich weniger Kohlenmonoxid produzieren“; ibid. p. 32). Trunk then goes on to mention in a footnote that some Belzec witnesses stated that the corpses were blue, suggesting that this would fit with an observation of people murdered using a Diesel engine, as their cause of death would have been a “combination of carbon monoxide poisoning (inner asphyxation) and deprivation of oxygen (outer asphyxation). However, the witnesses mentioning blue gas chamber corpses in connection with Belzec also made statements regarding the time required for the gassings that are irreconcilable with Trunk’s assertion that Diesel gassings would have required a considerably longer time than 20 minutes to carry out. Gerstein claimed that the victims in the gas chambers were still alive at the time the Diesel gassing engine was finally started, and that the subsequent gassing took 32 minutes, with “only a few” remaining alive after 28 minutes. Wilhelm Pfannenstiel, who supposedly witnessed the same gassing at Belzec as Gerstein, testified that the gassing took either some 12 minutes (Ernst Klee, Willi Dressen, Wolker Riess, “Schöne Zeiten” Judenmord aus der Sicht der Täter und Gaffer, 2nd ed., S. Fischer, Frankfurt am Main 1988, p. 221) or around 18 minutes (cf. C. Mattogno, Belzec, op.cit., p. 56). About the engine type Pfannenstiel made only vague statements (cf. ibid., p. 59). Karl-Afred Schluch (see below), who is the third Belzec witness to mention the color blue, testified that the gassings took only some 5-7 minutes; ZStL, 208 AR-Z 252/59, vol. 8, pp. 1512 (also quoted online: http://www.ess.uwe.ac.uk/genocide/browningfn5.htm ). Schluch did not specify the engine type. So much for the reliability of Trunk’s hypothetical Diesel gassing witnesses. It is worth noting that Trunk (ibid., p. 28) states that “The victims of carbon monoxide poisoning are as a rule to be recognized by the red coloration of the mucous membranes, as the carbon monoxide-loaded hemoglobin  with (and thus the blood in its entirety) has a cherry-red color.” (“Die Opfer einer Kohlenmonoxid-Vergiftung sind in der Regel an einer Rotfärbung der Schleimhäute zu erkennen, da das mit Kohlenmonoxid beladene Hämoglobin (und damit das Blut insgesamt) eine kirschrote Farbe hat.”). However, as shown in illustrations 1-4 and by the medical reports in section 4, the cherry-red discoloration is far from restricted to the mucous membranes.

[7] Friedrich Paul Berg, “The Diesel Gas Chambers: Myth Within a Myth”, The Journal of Historical Review, Vol. 5 No. 1 (Winter 1984), p. 15f.

[8] Namely S. Kaye, Handbook of Emergency Toxicology, 4th ed., C.C. Thomas, Springfield 1980; and C.J. Polson, R.N. Tattersall, Clinical Toxicology, Lippincott, Philadelphia 1969.

[9] W. Forth, D. Henschler, W. Rummel, K. Starke, Allgemeine und spezielle Pharmakologie und Toxikologie, 6th ed., Wissenschaftsverlag, Mannheim 1992.

[10] The Revisionist ,No. 2, 2004, pp. 159-164.

[11] Friedrich Paul Berg, “Blue Women on the Beach – and the False Toxicity of CO2 in Diesel Exhaust”; Online: http://www.nazigassings.com/Provan.html

[12] A. Ernst, J.D. Zibrak, “Carbon monoxide poisoning”, The New England Journal of Medicine, Vol. 339, Iss. 22 (November 1998), p. 1604.

[13] The Journal of Emergency Medicine, Vol. 1, 1984, p. 236.

[14] F. Homburger, J.A. Hayes, E.W. Pelikan, A guide to general toxicology (Karger continuing education series; vol. 5), Karger, Basel/Tokyo 1983, p. 48.

[15] Indications that the authors are referring to clinical cases in this paragraph can be found in the following sentences (Ibid, emphasis added): “Once exposure to carbon monoxide ceases, however, the circulatory concentrations begin to decrease. (…) Although the presentation of carbon monoxide poisoning is highly variable and depends on the patient (…) the severity of the clinical presentation generally correlates with the severity of the exposure. (…) Central nervous system symptoms and signs include headache, dizziness, emotional lability, confusion and convulsion. Respiratory symptoms include shortness of breath ranging from mild dyspnea on exertion to fainting… (…) Carbon monoxide poisoning may result in blisters or bullae over pressure areas but the classic cherry red color of the skin is rare. Focal neurological defects in 30% of survivors who arrive in the emergency room in coma.”

[16] Another example: In the article “Carbon monoxide intoxication: an updated review” by L.D. Prockop and R.I. Chichkova (in Journal of the Neurological Sciences, Vol. 262 No. 1-2 (November 2007), pp. 122-130) we read: “The classic cherry-red discoloration of the skin and cyanosis are rarely seen.” This sentence is however found in an article section headed “Clinical findings”, and again we can also glean from the context that the authors are referring to treated patients, for the following sentence reads: “Varying degrees of cognitive impairment have been reported”.

[17] D. Nicholas Bateman, “Carbon monoxide”, Medicine, Vol. 35, No. 11 (November 2007), pp. 605.

[18] Bruno Simini, “Cherry-red discolouration in carbon monoxide poisoning”, The Lancet, Vol. 352 (October 1998), p. 1154.

[19] Image found at http://www.acsu.buffalo.edu/~lcscott/carbonmonoxide.html (This as well as the two following illustrations were found and used by Friedrich Paul Berg in his rebuttal to Provan).

[20] Jay Dix, Forensic Pathology – A Color Atlas on CD-ROM, CRC Press, Boca Raton, p. 111.

[21] Forensic Medicine: Colour Guide, Churchill Livingstone, Edinburgh/New York 2003, p. 12.

[22] Textbook of Maritime Medicine: 10.9. Deaths on Board, online: http://textbook.ncmm.no/medical-challenges-on-board/501-claas-buschmann

[23] Bruce L. Danto, M.D., “The Man with a Red Face”, The American Journal of Psychiatry, Vol. 121:3 (September 1964), pp. 275-276. Cf. also John J. Miletich, Tia Laura Lindstrom, Cyril H. (FRW) Wecht, An Introduction to the Work of a Medical Examiner: From Death Scene to Autopsy Suite, ABC-CLIO, 2010, p. 16: “The blood of a person who died of carbon monoxide poisoning will continue to be bright red after death; the blood of someone who died of cyanide poisoning will be pink” (emphasis added); . This statement by Miletich clearly implies that the discoloration is a phenomenon in effect before death.

[24] A.F. Sedda, G. Rossi, “Death scene evaluation in a case of fatal accidental carbon monoxide toxicity”, Forensic Science International, Vol. 164, No. 2-3 (December 2006), pp. 164-167.

[25] P. Schmidt, F. Musshoff, R. Dettmeyer, B. Madea, “Unusual carbon monoxide poisoning”, Archiv für Kriminologie, Vol. 208 No. 1-2 (July-August 2001), pp. 10-23.

[26] H.J. Carson, K. Esslinger, “Carbon monoxide poisoning without cherry-red livor”, The American Journal of Forensic Medicine and Pathology, Vol. 22, No. 3 (September 2001), pp. 233-235.

[27] G.H. Findlay, “Carbon monoxide poisoning: optics and histology of skin and blood”, British Journal of Dermatology, Vol. 119 No. 1 (July 1988), pp. 45-51.

[28] S.R. Metha, M. Niyogi et al., “Carbon Monoxide Poisoning”, The Journal of the Association of Physicians of India, Vol. 49 (June 2001), pp. 622-625.

[29] Daniele Risser, Anneliese Bönsch, Barbara Schneider, “Should coroners be able to recognize unintentional carbon monoxide-related deaths immediately at the death scene?“, The Journal of Forensic Science, Vol. 40 No. 4 (July 1995), pp. 596-598.

[30] Livor mortis, also known as post mortem lividity or hypostasis, is an indicator of death. The term refers to the settling or pooling of blood in the lower portions of the body, causing purplish red discoloration of the skin. The state is due to red blood cells sinking through the serum (the liquid component of the blood) when the heart is no longer pumping the blood through the blood vessels. Due to capillary compression, discoloration does not appear in areas of the body that are in contact with the ground or other surfaces. For the time of the appearance of livor mortis, see below. When the authors of the article speak of a “cherry-pink coloring of livor mortis” they are referring to a discoloration of a nuance distinct from that normally characteristic of livor mortis.

[31] Ibid., p. 597.

[32] Ibid., p. 598.

[33] A.H. Thomsen, M. Gregersen, “Suicide by carbon monoxide from car exhaust-gas in Denmark 1995-1999”, Forensic Science International, Vol. 161, No. 1 (August 2006), pp.41-46.

[34]On jètes les corps, bleus humides soudre et de l’urine, les jambes pleins de crotte et de sangue périodique.“ (This is how the handwritten text (T I) reads; the typewritten text (T II) inserts a comma after the word bleus). H. Roques, The “Confessions“ of Kurt Gerstein, Institute for Historical Review, Costa Mesa 1989, p. 24, 32, 216, 225.

[35] Interrogation of Wilhelm Pfannenstiel on June 6, 1950, ZStL, 208 AR-Z 252/59, Vol. I, p. 44.

[36]Die Leichen waren wenigstens teilweise mit Kot und Urin, andere zum Teil mit Speichel besudelt. Bei den Leichen konnte ich z.T. sehen, dass die Lippen und auch Nasenspitzen blaulich verfärbt waren. Bei einigen waren die Augen geschlossen, bei anderen waren die Augen verdreht.”. ZStL, 208 AR-Z 252/59, vol. 8, pp. 1512-1513.

[37]Ich selbst wurde zu einer Art Waldwiese gefahren und als ich dort ankam, bog auch schon dieser Omnibus ein, er fuhr an eine ausgehobene Grube; die Türe wurde aufgemacht und heraus purzelten Leichen; in die Grube hinein. Eine über die andere. Das war ein schauriges Inferno. Nein, es war ein Superinferno. Eben sah ich sie noch lebendig. Nun waren sie samt und sonders tot.“ Quoted from “Manuscript of Adolf Eichmann’s Memoirs”, reportedly written in Haifa, Israel, in 1961, p. 127.

[38] R. Sakowska, Die zweite Etappe ist der Tod. NS-Ausrottungspolitik gegen die polnischen Juden gesehen mit den Augen der Opfer, Edition Entrich, Berlin 1993, s. 163, 166.

[39] Quoted in Carlo Mattogno, Bełżec, op.cit., p. 38.

[40] In the previously published version of this article Treblinka key witness Jacob (Jankiel) Wiernik was listed as witness number 5, due to the English (as well as Yiddish) translation of his pamphlet A Year in Treblinka mentioning “yellow” corpses (“There was no longer beauty or ugliness, for they all were yellow from the gas”, in the Polish original: “Nie ma ładnych i brzydkich, wszyscy żółci-zatruci.”). It has since been pointed out to us by a scholar who wishes to remain anonymous that we are here dealing with a mistranslation of a Polish idiomatic expression, żółci-zatruci, where “żółci” does not come from the word for “yellow” (żółty) but for “gall” (żółć) which has in vernacular an association with “poison”, cf. the German expression “Gift und Galle“. Thus Wiernik (in his known testimonies) has nothing concrete to say about the appearances of the corpses.

[41]Die Körper waren stark aufgedunsen, die Haut grau-weisslich und löste sich leicht,so dass sie oft in Fetzen herunterhing. Die Augen waren herabgequollen und die Zunge hing aus dem Mund.” Elias Rosenberg, “Tatsachenbericht“ signed in Vienna, December 12, 1947, p. 5; reproduced in H.P. Rullmann, Der Fall Demjanjuk – Unschuldiger oder Massenmörder?, Verlag Helmut Wild, 1987, p. 137; available online: http://www.vho.org/D/dfd/5.html

[42] Alexander Donat (Ed.), The Death Camp Treblinka: A Documentary, Holocaust Library, New York 1979, p. 36.

[43] David Mittelberg, Between Two Worlds: The Testimony & The Testament, Devora Publishing, Jerusalem/New York 2004, p. 44.

[44] Antony Beevor, Luba Vinogradova (eds.), A writer at war: Vasily Grossman with the Red Army, 1941-1945, Pantheon Books 2005, p. 298.

[45] Quoted in Eugen Kogon, Hermann Langbein, Adalbert Rückerl (eds.), Nationalsozialistische Massentötungen durch Giftgas, Frankfurt/M.: S. Fischer Verlag, 1983, p. 83f.

[46] Myron Winick (ed.), Hunger Disease. Studies by the Jewish Physicians in the Warsaw Ghetto, John Wiley & Sons, New York 1979.

[47] Ibid, pp. vii-ix.

[48] Ibid, pp. 29-30.

[49] Ibid, p. 30.

[50] Ibid, p. 30.

[51] Ibid, p. 53.

[52] Ibid, p. 63.

[53] Ibid, pp. 158-159.

[54] Ibid, p. 165.

[55] Ibid, p. 185.

[56] Ibid, pp. 190-191.

[57] Ibid, p. 233.

[58] Cf. Jürgen Graf, Thomas Kues, Carlo Mattogno, Sobibór. Holocaust Propaganda and Reality, TBR Books, Washington DC 2010, pp. 145-146.

[59] A. Harper, J. Croft-Baker, “Carbon monoxide poisoning: undetected by both patients and their doctors”, Age and Ageing, Vol. 33, No 2 (2004), p. 107.

[60] It should be noted that another characteristic sign of carbon monoxide poisoning is retinal hemorrhages, i.e. bleedings within the eye’s retina. As far the author is aware, this symptom, which would likewise be quite visible, has not been mentioned by any “gas chamber” eyewitness. Cf. R.A. Etzel, “The “fatal four” indoor air pollutants”, Pediatric Annals, Vol. 29, No. 6 (June 2000), p. 346.